High Triglycerides > Dietary Tips

Health Condition

High Triglycerides

The right diet is the key to managing many diseases and to improving general quality of life. For this condition, scientific research has found benefit in the following healthy eating tips.

  • High-Fiber Diet

    Diets high in fiber have reduced triglyceride levels in some studies. Water-soluble fibers, such as those found in fruit, beans, and oats, may be particularly helpful.
    High-Fiber Diet
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    Diets high in fiber have reduced TG levels in several clinical trials,14 but have had no effect in other clinical trials.15 Water-soluble fibers, such as pectin found in fruit, guar gum and other gums found in beans, and beta-glucan found in oats, may be particularly helpful in lowering triglycerides.

  • Dietary Caffeine

    In a study of heavy caffeine users, changing to decaffeinated coffee and eliminating all other caffeinated products reduced triglyceride levels by 25%.
    Dietary Caffeine
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    In a study of heavy caffeine users (individuals who were consuming an average of 560 mg of caffeine per day from coffee and tea), changing to decaffeinated coffee and eliminating all other caffeinated products for two weeks resulted in a statistically significant 25% reduction in TG levels.16

  • Fish

    Triglyceride-lowering omega-3s are found in fatty fish such as herring, mackerel, sardines, anchovies, albacore tuna, and black cod.
    Fish
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    Some,17,18 but not all,19 studies have found that increasing consumption of fish is associated with a lower risk of heart disease. Significant amounts of TG-lowering omega-3 fatty acids (EPA and DHA) can be found in the fish oil of salmon, herring, mackerel, sardines, anchovies, albacore tuna, and black cod. Many doctors recommend that people with elevated TGs increase their intake of these fatty fish.

  • Sugar

    People with elevated triglycerides should replace sugary foods and beverages with natural, unsweetened options, as refined sugar increases triglyceride levels.
    Sugar
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    Ingesting refined sugar increases TG levels, as well.20,21 People with elevated TGs should therefore reduce their intake of sugar, sweets, and other sugar-containing foods. There is also evidence that ingesting fructose in amounts that are found in a typical Western diet can raise TG levels, although not all studies agree on that point.22 It should be noted that most studies of fructose investigated the refined form, not the fructose that occurs naturally in some fruits.

  • Unsaturated Fats

    Many doctors recommend a diet higher in unsaturated fats, such as olive oil, over a diet high in saturated fat to reduce triglycerides and heart disease risk. In other words, choose fish, soy, and nonfat dairy, and avoid meats and fatty dairy.
    Unsaturated Fats
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    The blood level of TGs following a meal may be a more important indicator of coronary heart disease risk than the fasting level.23,24 However, a low-fat diet (55% carbohydrates, 23% fats, 22% proteins) that succeeded in normalizing other blood lipids, including fasting TG levels, failed to normalize post-meal TG levels in a group of people with hypertriglyceridemia.25 These results suggest that dietary reduction of fasting TGs, even if the diet controls other blood lipids, may not be enough to provide optimal protection against coronary heart disease. Many doctors recommend a diet low in saturated fat (meaning avoidance of red meat and all dairy except nonfat dairy) to reduce TGs and the risk of heart disease.26

  • Low-Fat, High-Carbohydate

    In one study, a low-fat diet high in unrefined carbohydrates reduced triglycerides.
    Low-Fat, High-Carbohydate
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    Consumption of a low-fat, high-carbohydrate diet reduced TGs in one study.27 However, in another study, populations that consumed a low-fat, high-carbohydrate diet had higher TG levels, compared with populations that consumed lower amounts of carbohydrates.28 Suddenly switching to a high-carbohydrate, low-fat diet will generally increase TGs temporarily, but making the switch gradually protects against this short-term problem.29

  • Alcohol Consumption

    While drinking moderate amounts of alcohol does not appear to affect triglyceride, heavy drinking is believed to increase levels.
    Alcohol Consumption
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    While consuming moderate amounts of alcohol does not appear to affect TG levels, heavy drinking is believed to be an important cause of hypertriglyceridemia.30 Alcoholics with elevated TG levels should deal with the disease of alcoholism first.

References

1. Prichard BN, Smith CCT, Ling KLE, Betteridge DJ. Fish oils and cardiovascular disease. BMJ 1995;310:819-20 [editorial/review].

2. Von Schacky C, Fischer S, Weber PC. Long-term effects of dietary marine omega-3 fatty acids upon plasma and cellular lipids, platelet function, and eicosanoid formation in humans. J Clin Invest 1985;76:1626-31.

3. Leaf A, Weber PC. Cardiovascular effects of n-3 fatty acids. N Engl J Med 1988;318:549-57 [review].

4. Adler AJ, Holub BJ. Effect of garlic and fish-oil supplementation on serum lipid and lipoprotein concentrations in hypercholesterolemic men. Am J Clin Nutr 1997;65:445-50.

5. Haglund O, Luostarinen R, Wallin R, et al. The effects of fish oil on triglycerides, cholesterol, fibrinogen and malondialdehyde in humans supplemented with vitamin E. J Nutr 1991;121:165-9.

6. Oostenbrug GS, Mensink RP, Hornstra G. A moderate in vivo vitamin E supplement counteracts the fish-oil-induced increase in in vitro oxidation of human low-density lipoproteins. Am J Clin Nutr 1993;57:827S.

7. Agarwal RC, Singh SP, Saran RK, et al. Clinical trial of gugulipid new hypolipidemic agent of plant origin in primary hyperlipidemia. Indian J Med Res 1986;84:626-34.

8. Arsenio L, Bodria P, Magnati G, et al. Effectiveness of long-term treatment with pantethine in patients with dyslipidemia. Clin Ther 1986;8:537–45.

9. Avogaro P, Bon B, Fusello M. Effect of pantethine on lipids, lipoproteins and apolipoproteins in man. Curr Ther Res 1983;33;488-93.

10. Maggi GC, Donati C, Criscuoli G. Pantethine: a physiological lipomodulating agent, in the treatment of hyperlipidemias. Curr Ther Res 1982;32:380-6.

11. Brown WV. Niacin for lipid disorders. Postgrad Med 1995;98:183-93 [review].

12. Head KA. Inositol hexaniacinate: a safer alternative to niacin. Alt Med Rev 1996;1:176-84 [review].

13. Murray M. Lipid-lowering drugs vs. Inositol hexaniacinate. Am J Natural Med 1995;2:9-12 [review].

14. Anderson JW, Gustafson NJ. High-carbohydrate, high-fiber diet. Postgrad Med 1987;82:40-55 [review].

15. Glore SR, Van Treeck D, Knehans AW, Guild M. Soluble fiber and serum lipids: a literature review. J Am Dietet Assoc 1994;94:425-36.

16. Naismith DJ, Akinyanju PA, Szanto S, Yudkin J. The effect in volunteers of coffee and decaffeinated coffee on blood glucose, insulin, plasma lipids and some factors involved in blood clotting. Nutr Metab 1970;12:144-51.

17. Burr ML, Fehily AM, Gilbert JF, et al. Effects of changes in fat, fish, and fibre intakes on death and myocardial reinfarction: diet and reinfarction trial (DART). Lancet 1989;ii:757-61.

18. Kromhout D, Bosschieter EB, de Lezenne Coulander C. The inverse relation between fish consumption and 20-year mortality from coronary heart disease. N Engl J Med 1985;312:1205-9.

19. Ascherio A, Rimm EB, Stampfer MJ, et al. Dietary intake of marine n-3 fatty acids, fish intake, and the risk of coronary disease among men. N Engl J Med 1995;332:977-82.

20. Reiser S. Effect of dietary sugars on metabolic risk factors associated with heart disease. Nutr Health 1985;3:203-16.

21. Szanto S, Yudkin J. The effect of dietary sucrose on blood lipids serum insulin, platelet adhesiveness and body weight in human volunteers. Postgrad Med J 1969;45:602-7.

22. Hollenbeck CB. Dietary fructose effects on lipoprotein metabolism and risk for coronary artery disease. Am J Clin Nutr 1993;58(5 Suppl):800S-9S.

23. Patsch JR, Miesenbock G, Hopferwieser T, et al. Relation of triglyceride metabolism and coronary artery disease. Studies in the postprandial state. Arterioscler Thromb 1992;12:1336-45.

24. Ryu JE, Howard G, Craven TE, et al. Postprandial triglyceridemia and carotid atherosclerosis in middle-aged subjects. Stroke 1992;23:823-8.

25. Zoppo A, Maggi FM, Catapano AL. A successful dietary treatment fails to normalize plasma triglyceride postprandial response in type IV patients. Atherosclerosis 1999;146:19-23.

26. Consensus Development Panel. Treatment of hypertriglyceridemia. JAMA 1984;251:1196-200.

27. Cominacini L, Zocca I, Garbin U, et al. Long-term effect of a low-fat, high carbohydrate diet on plasma lipids of patients affected by familial endogenous hypertriglyceridemia. Am J Clin Nutr 1988;48:57-65.

28. West C, Sullivan DR, Katan MB, et al. Boys from populations with high-carbohydrate intake have higher fasting triglyceride levels than boys from populations with high-fat intake. Am J Epidemiol 1990;131:271-82.

29. Ullmann D, Connor WE, Hatcher LF, et al. Will a high-carbohydrate, low-fat diet lower plasma lipids and lipoproteins without producing hypertriglyceridemia? Arterioscler Thromb 1991;11:1059-67.

30. Steinberg D, Pearson TA, Kuller LH. Alcohol and atherosclerosis. Ann Intern Med 1991;114:967-76.

31. Merrill JR, Holly RG, Anderson RL, et al. Hyperlipemic response of young trained and untrained men after a high fat meal. Arteriosclerosis 1989;9:217-23.

32. Cowan LD, Wilcosky T, Criqui MH, et al. Demographic, behavioral, biochemical, and dietary correlates of plasma triglycerides. Arteriosclerosis 1985;5:466-80.

33. Despres J-P, Tremblay A, Leblanc C, Bouchard C. Effect of the amount of body fat on the age-associated increase in serum cholesterol. Prev Med 1988;17:423-31.